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Joined 1 year ago
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Cake day: August 13th, 2024

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  • Congress, you have a choice, assert your powers, you know the ones you have had since November 7, 1973. Or give in, give up, and go home. Your constituents have put you in the position, as a co-equal branch, to restrain The Executive Branch. Do your JOB!

    https://en.wikipedia.org/wiki/War_Powers_Resolution

    "The War Powers Resolution (also known as the War Powers Resolution of 1973 or the War Powers Act) (50 U.S.C. ch. 33) is a federal law intended to check the U.S. president’s power to commit the United States to an armed conflict without the consent of the U.S. Congress. The resolution was adopted in the form of a United States congressional joint resolution. It provides that the president can send the U.S. Armed Forces into action abroad only by Congress’s “statutory authorization”, or in case of “a national emergency created by attack upon the United States, its territories or possessions, or its armed forces”. "


  • https://www.epa.gov/indoor-air-quality-iaq/nitrogen-dioxides-impact-indoor-air-quality#Health_Effects

    Health Effects Associated with Nitrogen Dioxide

    https://cfpub.epa.gov/ncea/iris/iris_documents/documents/subst/0080_summary.pdf

    NO2 acts mainly as an irritant affecting the mucosa of the eyes, nose, throat and respiratory tract.
    Extremely high-dose exposure (as in a building fire) to NO2 may result in pulmonary edema and diffuse lung injury.
    Continued exposure to high NO2 levels can contribute to the development of acute or chronic bronchitis.
    Low level NO2 exposure may cause:
        increased bronchial reactivity in some asthmatics
        decreased lung function in patients with chronic obstructive pulmonary disease
        increased risk of respiratory infections, especially in young children
    

    Home interventions are effective at decreasing indoor nitrogen dioxide concentrations https://pmc.ncbi.nlm.nih.gov/articles/PMC4909253/

    Abstract

    Nitrogen dioxide (NO2), a by-product of combustion produced by indoor gas appliances such as cooking stoves, is associated with respiratory symptoms in those with obstructive airways disease. We conducted a three-armed randomized trial to evaluate the efficacy of interventions aimed at reducing indoor NO2 concentrations in homes with unvented gas stoves: (i) replacement of existing gas stove with electric stove; (ii) installation of ventilation hood over existing gas stove; and (iii) placement of air purifiers with high-efficiency particulate air (HEPA) and carbon filters. Home inspection and NO2 monitoring were conducted at 1 week pre-intervention and at 1 week and 3 months post-intervention. Stove replacement resulted in a 51% and 42% decrease in median NO2 concentration at 3 months of follow-up in the kitchen and bedroom, respectively (P = 0.01, P = 0.01); air purifier placement resulted in an immediate decrease in median NO2 concentration in the kitchen (27%, P < 0.01) and bedroom (22%, P = 0.02), but at 3 months, a significant reduction was seen only in the kitchen (20%, P = 0.05). NO2 concentrations in the kitchen and bedroom did not significantly change following ventilation hood installation. Replacing unvented gas stoves with electric stoves or placement of air purifiers with HEPA and carbon filters can decrease indoor NO2 concentrations in urban homes.

    I find it interesting when Judges, such as Justice S. Kato Crews, appear unable to do literature searches, and/or appear unable to read said literature.

    From OP’s article, this pertinent review: Review from American Lung Association: https://www.lung.org/getmedia/ce1e7bfa-db92-42a3-9ff4-3044324ec2c9/ICF_Impacts-of-Residential-Combustion_Addendum.pdf












  • Right, so all of this indicates that phytoestrogens indeed do have effects on mice and human estrogen related physiology. The effects can be a little puzzling, though. As I understand it, there are two types of estrogen receptors, Alpha and Beta. And there are many phytoestrogens as well as estrogenic chemicals that we are exposed to, with the one’s in beer binding to Estrogen Receptor Alpha, which signals breast cells and breast cancer cells to proliferate. The binding affinity of the phytoestrogens is weaker than that of the endogenous estrogen. None the less when a given phytoestrogen is bound to a given receptor it gives a slightly weaker estrogen-like signal, and simultaneously prevents the binding of the endogenous estrogen (can’t fit two keys into a single lock at the same time), which has a stronger affinty, and thus stronger effect when it is bound to the receptor. So in an individual with lots of estrogen, the phytoestrogen may lower the estrogenic signal, and in someone with less estrogen it may augment the estrogenic signal in the body. Does that make sense? Do you have questions?